i(12p)
Pallister-Killian
syndrome
Epidemiology and
Etiology:
- rare
- Mosaic
form usually
- Tetrasomy for 12p
- Advanced
maternal age is a risk factor
- Mechanisms:
- Centromeric misdivision
at meiosis I
- Premeiotic centromere
misdivision
- Nondisjunction at meiosis I
- Leading
to a trisomic zygote
- Followed
by post-mitotic centromeric misdivision
- Premeiotic nondisjunction
- Giving
rise to a trisomic cell
- Followed
by centromeric misdivision
Common sites:
- Blood
is usually negative
Gross features:
- Wide
phenotypic variability
- Facies:
- “coarse”
features
- Prominent
forehead
- Sparse
hair or temporal alopecia
- Hypertelorism
- Flat
nasal bridge
- Short
nose with anteverted nares
- macrostomia
- Short
neck
- Cleft
lip / palate
- Pigmentary dysplasia
- Streaks
of hypo/hyper-pigmentation and facial anomalies
- Short
and broad hands
- Diaphragmatic
hernia
- Congenital
heart defects
- Anorectal anomalies
- Genital
malformations
- Supernumerary
nipples
Histologic
features:
Immunophenotype:
Marker:
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Sensitivity:
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Specificity:
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Molecular features:
Other features:
- Nearly
100% risk of fetal abnormality
- variable
phenotype, but usually very severe:
- Prenatal
death due to multiple congenital anomalies
- Severe
mental retardation (almost all)
- Seizures
(~60%)
- Hypotonia (almost all)
- U/S:
- Hydramnios
- Congenital
diaphragmatic hernia
- Micromelia (predominantly rhizomelic type)
- Less
common:
- Hydrops fetalis
- Increased
nuchal translucency (INT)
- Hygroma coli
- Fetal
overgrowth
- Ventriculomegaly
- Dilation
of the cavum pellicidum
- Absence
of stomach visualization
- Sacral
appendix
- Blood
samples negative usually
- Lymphocyte
karyotype is usually normal
- i(12p) found in skin
fibroblasts, amniocytes, and bone marrow cells
References:
- Gardner
RJM, Sutherland GR. Chromosome Abnormalities and Genetic Counseling. 2nd
ed. Oxford University
Press, USA; 1996.
- Genevieve
D et al. Mild Phenotype in a
15-Year-old boy with Pallister-Killian
Syndrome. Am J Med Genet. 2003;116A:90-93.